Robert Mentzer, MD
- The doctor practices in an office or specialty that we currently do not survey.
- The doctor has not yet received the minimum number of patient satisfaction surveys (30) to be eligible for display.
- The doctor practices in an office or specialty that we currently do not survey.
- The doctor has not yet received the minimum number of patient satisfaction surveys (30) to be eligible for display.
Robert Mentzer, MD
Los Angeles
127 S San Vicente Blvd #A9100
Los Angeles, CA
34.0746919 -118.3789723From You
- The doctor practices in an office or specialty that we currently do not survey.
- The doctor has not yet received the minimum number of patient satisfaction surveys (30) to be eligible for display.
Robert Mentzer, MD
Languages
- English,
Gender
Male
Experience
51 Years
Locations
Experience
Specialties
Area in which a healthcare provider is highly trained and often board certified.
1
- Research
Programs
Research Areas
The research interests of Robert M Mentzer Jr., MD, include myocardial ischemia/reperfusion injury, myocardial protection, adverse post-infarction left ventricular remodeling, heart failure and organ preservation for heart transplantation. His studies focus on mitophagy and mitochondrial biogenesis and the role these play in the mitigation of acute and chronic ischemic injury. The objective is to develop more effective cardioprotective strategies for patients at risk of ischemia-reperfusion injury and heart failure using an entirely new approach to cardioprotection based on adaptive autophagy.
Titles
- Professor, Surgery
- Professor, Medicine
Education & Training
-
University of Virginia Medical Center
Medical School - Completed 1971
-
University of Maryland
Degrees - Completed 1967
Achievements
-
6
Publications
Publications
A full list of this provider’s research publications can be found on their research profile or PubMed.
- Andres AM, Germano J, Huang C, Tucker KC, Song Y, Sin J, Gottlieb RA, and Mentzer RM. Pioglitazone and Glucagon-like Peptide 1 Receptor Agonist Stimulate Mitochondrial Turnover in the Heart When Administered After the Infarct in Obese Mice.. Circulation Research. 2019;123:A523
- Piplani H, Sharma A, Huang C, Sin J, Germano J, Mentzer RM, Andres AM, and Gottlieb RA. Asporin - Extracellular Matrix Protein Mitigates Pathological Remodeling. Circulation Research, 2019:123: A529 Germano J, Huang C, Song Y, Robert M Mentzer RM, Gottlieb RA, and Andres AA. GLP-1 Receptor Agonist Administered After the Infarct Suppresses miR-33 Expression in Lean Mice Subjected to Permanent Coronary Artery Occlusion. Circulation Research 2019; 123:A398
- Andres AM, Germano J, Huang C, Sin J, Song Y, Tucker KC, Gottlieb RA, and Mentzer RM. Glucagon-Like Peptide 1 Receptor Agonist Mitigates Postinfarction Adverse Left Ventricular (LV) Remodeling in Mice; a Mechanism Linked to Mitophagy and the Restoration of Cardiac Fatty Acid Oxidation. Originally published 5 Nov 2018 Circulation.2018;138:A16932
- Andres AM, Song Y, Gottlieb RA, and Mentzer RM. Empagliflozin Directly Stimulates PGC-1α-Dependent Mitochondrial Biogenesis in Cardiac Cells; A Potential Mechanism Associated With Its Cardiovascular Benefits. Originally published 5 Nov 2018 Circulation. 2018;138:A16629
- Andres, AM, Tucker, KC, Thomas A, Taylor DJ, Sengstock, D, Jahania, S. M., Dabi, R, Pourpirali, S., Brown, JA, Westbrook, DG, Ballinger, S. W., Mentzer, RM, Gottlieb, R. A. (2017). Mitophagy and mitochondrial biogenesis in atrial tissue of patients undergoing heart surgery with cardiopulmonary bypass. JCI Insight, 2017:2(4), e89303. doi:10.1172/jci.insight.89303 PMID:2823965
- Recent Publications: Andres AM, Kooren JA, Parker SJ, Tucker KC, Ravindran N, Ito BR, Huang C, Venkatraman V, Van Eyk JE, Gottlieb RA, Mentzer RM Jr. Discordant signaling and autophagy response to fasting in hearts of obese mice: Implications for ischemia tolerance. Am J Physiol Heart Circ Physiol. 2016 Jul 1;311(1):H219-28. PMID:2719911Giricz Z, Varga, ZV, Koncsos G., Nagy, CT, Görbe, A., Mentzer RM, Gottlieb, RA, Ferdinandy, P. (2017). Autophagosome formation is required for cardioprotection by chloramphenicol. Life sciences, 2017.186, 11-16. PMID:28778689