
Carlos J. Gonzalez Quesada, MD, MPH
- The doctor practices in an office or specialty that we currently do not survey.
- The doctor has not yet received the minimum number of patient satisfaction surveys (30) to be eligible for display.
310-278-3400
Fax: 310-278-1240

- The doctor practices in an office or specialty that we currently do not survey.
- The doctor has not yet received the minimum number of patient satisfaction surveys (30) to be eligible for display.
Carlos J. Gonzalez Quesada, MD, MPH
Fax: 310-278-1240
Beverly Hills
414 N Camden Dr
Ste 1100
Beverly Hills, CA
34.0690005 -118.4039646From You
- The doctor practices in an office or specialty that we currently do not survey.
- The doctor has not yet received the minimum number of patient satisfaction surveys (30) to be eligible for display.

Carlos J. Gonzalez Quesada, MD, MPH
Languages
- English,
- Spanish,
- Tagalog,
- Chinese (Mandarin),
Gender
Male
Experience
15 Years
Locations
Experience
Specialties
Area in which a healthcare provider is highly trained and often board certified.
1
- IM Cardiology
Research Areas
Education & Training
-
Alliance for Physician Certification & Advancement, Certification Board of Cardiovascular Computed Tomography
Education - Completed 2028
-
National Board of Echocardiography, Comprehensive Certification in Adult Echocardiography
Education - Completed 2027
-
Mayo Clinic, Structural Cardiology
Fellowship - Completed 2020
-
Mayo Clinic, Interventional Cardiology
Fellowship - Completed 2019
-
UCLA/Ronald Reagan UCLA Medical Center, Cardiology
Fellowship - Completed 2018
-
Harvard Medical School/Brigham and Women's Hospital, Global Health Equity
Residency - Completed 2015
-
Harvard Medical School/Brigham and Women's Hospital, Internal Medicine
Residency - Completed 2015
-
Harvard School of Public Health
Degrees - Completed 2015
-
Albert Einstein College of Medicine of Yeshiva University, Postdoctoral fellowship in cardiovascular diseases
Education - Completed 2011
-
Baylor College of Medicine, Cardiovascular Sciences
Education - Completed 2010
-
Universidad De La Salle/Mexico City
Medical School - Completed 2007
-
Alliance for Physician Certification & Advancement, Certification Board of Nuclear Cardiology
Education
-
Alliance for Physician Certification & Advancement, Physicians' Vascular Interpretation
Education
Achievements
-
3
Board Certifications
-
20
Publications
Board Certifications
-
General Internal Medicine
American Board of Internal Medicine
-
IM/Cardiology
American Board of Internal Medicine
-
Interventional Cardiology
American Board of Internal Medicine
Publications
- Knot in the Right Place
- Smad3 Signaling Promotes Fibrosis While Preserving Cardiac and Aortic Geometry in Obese Diabetic Mice
- Selecting an Oral Anticoagulant for Patients With Nonvalvular Atrial Fibrillation
- CXCR3-independent Actions of the CXC Chemokine CXCL10 in the Infarcted Myocardium and in Isolated Cardiac Fibroblasts Are Mediated Through Proteoglycans
- Comparison of the Phase III Clinical Trial Designs of Novel Oral Anticoagulants Versus Warfarin for the Treatment of Nonvalvular Atrial Fibrillation: Implications for Clinical Practice
- Computed Tomography and Echocardiography in Patients With Acute Pulmonary Embolism: Part 2: Prognostic Value
- Computed Tomography and Echocardiography in Patients With Acute Pulmonary Embolism: Part 1: Correlation of Findings of Right Ventricular Enlargement
- Thrombospondin-1 Induction in the Diabetic Myocardium Stabilizes the Cardiac Matrix in Addition to Promoting Vascular Rarefaction Through angiopoietin-2 Upregulation
- Thrombospondin-1 Regulates Adiposity and Metabolic Dysfunction in Diet-Induced Obesity Enhancing Adipose Inflammation and Stimulating Adipocyte Proliferation
- Systematic Characterization of Myocardial Inflammation, Repair, and Remodeling in a Mouse Model of Reperfused Myocardial Infarction
- Endogenous Thrombospondin 1 Protects the Pressure-Overloaded Myocardium by Modulating Fibroblast Phenotype and Matrix Metabolism
- Smad3 Signaling Critically Regulates Fibroblast Phenotype and Function in Healing Myocardial Infarction
- CCR5 Signaling Suppresses Inflammation and Reduces Adverse Remodeling of the Infarcted Heart, Mediating Recruitment of Regulatory T Cells
- Induction of the CXC Chemokine Interferon-Gamma-Inducible Protein 10 Regulates the Reparative Response Following Myocardial Infarction
- The Extracellular Matrix as a Modulator of the Inflammatory and Reparative Response Following Myocardial Infarction
- Monocyte Chemoattractant protein-1/CCL2 as a Biomarker in Acute Coronary Syndromes
- Left Main Coronary Artery Stenosis Treatment With Two Paclitaxel-Eluting Stents in a Patient With Cardiac Allograft Vasculopathy
- No Association Found Between the I/D of a 287-bp Alu Repeat Sequence Within Intron 16 of the ACE Gene in Mexican Patients and Binary Restenosis After Coronary Stenting
- The Interleukin 1B-511 Polymorphism Is Associated With the Risk of Developing Restenosis After Coronary Stenting in Mexican Patients
- Predictors of Mortality and Adverse Outcome in Elderly High-Risk Patients Undergoing Percutaneous Coronary Intervention